Skip to main content
. 2011 Nov;32(10):1771–1775. doi: 10.3174/ajnr.A2265

Fig 2.

Fig 2.

FVH is often transient and is a marker of collateral flow. A, In case 1 (top row), preangiogram FLAIR image demonstrates FVH in the left temporal lobe. B, The angiogram obtained soon after demonstrates MCA occlusion and very good leptomeningeal collateral flow. The MR image obtained at 24 hours after presentation (C) demonstrates absence of FVH, the presence of T2 signal-intensity abnormality in the basal ganglia and subcortical white matter consistent with acute infarction, and a normal flow-related enhancement in the left MCA on a concurrent MR angiogram (D). The absence of FVH on follow-up imaging is consistent with restoration of normal flow. In case 2 (bottom row), preangiogram FLAIR image (E) demonstrates FVH in the left temporal lobe, and an angiogram (F) obtained soon after shows MCA occlusion and very good collateral flow. G, MR image obtained the day after demonstrates continued presence of FVH and T2 signal-intensity abnormality in the basal ganglia, consistent with acute infarction. There is reduced flow-related enhancement in the left MCA territory on a concurrent MR angiogram (D), indicating continued abnormal arterial flow.