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. 2011 Aug;32(7):1321–1327. doi: 10.3174/ajnr.A2464

Table 1:

Reported location of hypoglycemic lesions based on DWI imaging of the cerebrum

Authors Patient (Age [yr], Sex) Blood Glucose (mmol/L) Symptoms Hyperintense Lesion Localization on DWI Reported Time between Onset of Hypoglycemic Symptoms and Initial DWI (hr)
Albayram et al7 68, F 1.77 Hemiparesis, slurred speech Internal capsules 1
Maekawa et al16 62, M 2.0 Comatose Cortical occipital lobe, hippocampus 18
Lo et al15 65, M 2.11 Seizures Diffuse cortical lesions 120
69, M 1.9 Speech difficulty, weakness Corona radiata, internal capsules, splenium of the corpus callosum 0.75
Kim et al14 78, F 2.10 Dysarthric, less responsive Splenium of the corpus callosum, internal capsules 2
Yanagawa et al18 20, F 3.69 Cortical and subcortical (sparing motor, sensory, and visual centers) 120
Kim et al13 78, F 2.1 Somnolent Splenium of the corpus callosum, internal capsules 2
41, M 1.16 Drowsy Splenium of the corpus callosum 2
42, M 0.16 Comatose Splenium of the corpus callosum 3
Yoneda and Yamamoto19 37, M n.a. Deeply comatose Periventricular white matter 24
Cortical frontal lobe and insula 504
Cordonnier et al11 24, F 2.2 Hemiparesis Internal capsula 2
Shirayama et al17 58, M 0.9 Deeply comatose Pons 3
Böttcher et al9 77, M 1.71 Hemiparesis, somnolent Corona radiata, splenium of the corpus callosum 1
Aoki et al8 73, F 1.1 Unresponsive Bilateral internal capsules, corona radiata, cortical frontal and parietal lobes On admission
Chan et al10 53, M 1.1 Seizures, diaphoresis Cortical, temporal, and occipital lobes 24
Finelli12 53, M 1.27 Unresponsive Basal ganglia, hippocampus, cortical temporal lobe 120