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. 2021 Mar 16;4:342. doi: 10.1038/s42003-021-01870-3

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 4 — a Recovery of delayed-type hypersensitivity in mouse footpad by J-1012 N-glycan. b, c Analyses of cytokine production by splenocytes from mice after 2 weeks of sepsis induction with i.v injection of OVA, J-1012 N-glycan and LPS without (b) or with anti-IL-10 mAb (c). The splenocytes were restimulated as described for 4 days. d Expression of CD62L and PD-1 of transferred DO11.10 T cells (CD4⁺KJ1-26⁺) (Supplementary Fig. 10) from mice after 2 weeks of sepsis induction with OVA, LPS and J-1012 N-glycan. The experiment was repeated twice, and compiled results are shown (right panels) (n = 6). a–c Data are expressed as the mean ± SD and are representative of at least two independent experiments (n = 3). p value was determined by unpaired two-tailed Student’s t-test. N.S., not significant (p > 0.05). R.IgG, control rat IgG. SRBC sheep red blood cells.