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. Author manuscript; available in PMC: 2022 Feb 1.
Published in final edited form as: Psychol Addict Behav. 2021 Feb;35(1):52–61. doi: 10.1037/adb0000595

Table 3.

Indirect effects from mediation analyses (0 = abstinent, 1 = relapsed)

95% CI†
Path B SE Upper Lower
P ➔ DM➔ SR −0.47 0.36 −1.31 −0.06
P ➔ PS➔SR −0.26 0.28 −1.00 0.06
P ➔ A➔ SR −0.16 0.18 −0.63 0.11
P ➔ SE ➔ SR1 0.01 0.26 −0.53 0.56
P ➔ SE ➔ SR2 −0.01 0.28 −0.52 0.57
P ➔ SE ➔ SR3 0.06 0.22 −0.36 0.55
P ➔PS ➔ SE ➔ SR 0.22 0.17 0.03 0.65
P ➔ DM ➔ SE ➔ SR 0.15 0.13 0.00 0.47
P ➔ A ➔ SE ➔ SR 0.06 0.06 −0.04 0.21

P = pain (How much bodily pain have you had during the past four weeks); DM = depressed mood (I feel depressed); PS = stress (I feel stressed); A = anxiety (I feel anxious), SE, mood-related smoking outcome expectancy (I am confident that smoking would improve my mood); SR = smoking relapse (based on biochemically confirmed 7-day point-prevalence smoking abstinence).

†

Bias corrected bootstrapped confidence intervals (10000 bootstrap samples).

1

This indirect pathway is based on the model that regressed smoking relapse on pain, stress, and mood-related smoking outcome expectancy.

2

This indirect pathway is based on the model that regressed smoking relapse on pain, depressed mood, and mood-related smoking outcome expectancy.

3

This indirect pathway is based on the model that regressed smoking relapse on pain, anxiety, and mood-related smoking outcome expectancy.

Note: Bolded text indicates a significant indirect effect.