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. 2021 Mar 17;12(4):284. doi: 10.1038/s41419-021-03561-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — A Western-blot showed shRNA-Rab26-1 efficiently depleted Rab26 in MCF7 cells. B Wound-healing assays showed Rab26 knockdown promotes migration of MCF7 cells. C Quantitative analysis of the wound area using imageJ software from three independent wound-healing experiments, *p < 0.05. D RTCA matrigel invasion assays showed Rab26 knockdown promotes invasion of MCF7 cells. **p < 0.01. E Double-layer soft agar experiments show that Rab26 knockdown promotes colony formation of MCF7 cells in soft agars. F Quantitative analysis of the results of E from three independent experiments. **p < 0.01.