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. 2021 Mar 17;19:112. doi: 10.1186/s12967-021-02709-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 5 — The interaction between PRDM1 and c-FOS facilitates the malignant behaviors of BCa cells. a The targeting relationship between PRDM1 and c-FOS verified by hTFtarget. b The expression of PRDM1 determined by RT-qPCR. c The expression of PRDM1 determined by Western blot analysis. d The expression of c-FOS determined by RT-qPCR. e The expression of c-FOS determined by Western blot analysis. f The proliferation of BCa cells detected by CCK-8 assay. g The apoptosis of BCa cells detected by TUNEL staining (×200). h The number of migratory and invasive BCa cells detected by Transwell assay (×200). i migration of BCa cells detected by scratch test (×200). *p < 0.05 vs. T24 cells treated with oe-NC. ^#p < 0.05 vs. T24 cells treated with sh-NC. ^&p < 0.05 vs. T24 cells treated with oe-PRDM1