fig 3.

Images obtained at the 1-year follow-up examination.

A, and B, Axial view spin-echo T2-weighted sequence. High intense signal is present in the gray and white matter of the right frontal lobe and of both parietal lobes. Note that compared with figure 1, there is progression of the disease in the left parietal lobe. The right parietal lobe, on the contrary, looks less involved.

C, Axial view spin-echo T2-weighted sequence. The lesion on the left is involving also the temporal lobe.

D, Axial view fluid-attenuated inversion recovery sequence obtained at the same level as that shown in A. The parietal lobes are diffusely hyperintense. High intensity signal is also present in the right frontal lobe. The right parietal lobe appears less involved than on the previous MR image.

E, Axial view fluid-attenuated inversion recovery sequence obtained at the same level as that shown in B. The parietal lobes are diffusely hyperintense. High intensity signal is also present in the right frontal lobe. The right parietal lobe appears less involved than on the previous MR image.

F, Axial view fluid-attenuated inversion recovery sequence obtained at the same level as that shown in C. The parietal lobes are diffusely hyperintense. High intensity signal is also present in the right frontal lobe. The right parietal lobe appears less involved than on the previous MR image.

G, Infused axial view spin-echo T1-weighted sequence. There is no evidence of abnormal contrast enhancement in the diseased regions.

H, Infused axial view spin-echo T1-weighted sequences. There is no evidence of abnormal contrast enhancement in the diseased regions.