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. 2021 Mar 19;16:137. doi: 10.1186/s13023-021-01770-z

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — Images of patient A diagnosed with LICS: a facial appearance. b Chest X-ray on day of admission showing bilateral interstitial infiltrates. c corresponding chest CT scan showing bilateral ground glass haziness, interlobar thickening and pneumomediastinum. e Lung tissue: overview showing pathological pattern of classic diffuse alveolar damage with extensive thickening of alveolar septa with early interstitial fibrosis (hematoxylin and eosin, scale bar 1 mm). g Lung tissue: Magnification: more detailed view, in which remnants of alveoli can be discerned with hyaline membranes, extensive metaplastic squamous alveolar epithelium and widened alveolar septa with variable inflammation and variable stages of fibrosis (hematoxylin and eosin, scale bar 200 micron)