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. 2021 Mar 8;12:650922. doi: 10.3389/fendo.2021.650922

Table 4.

COX regression analysis of different genetic models.

Genotype χ2 HR (95%CI) P
rs12696304 Codominance GG 1.0(ref.)
CC 8.16 3.35(1.46–7.66) 0.004
CG 8.96 2.65(1.40–5.02) 0.003
Dominance GG 1.0(ref.)
CC+CG 10.82 2.79(1.52–5.16) 0.001
Recessive GG+CG 1.0(ref.)
CC 0.071
rs10936599 Codominance TT 1.0(ref.)
CC 11.1 4.98(1.94–12.79) 0.001
TC 6.95 3.27(1.36–7.89) 0.008
Dominance TT 1.0(ref.)
CC+TC 9.05 3.72(1.58–8.74) 0.003
Recessive TT+TC 1.0(ref.)
CC 5.9 2.07(1.15–3.72) 0.015
rs1317082 Codominance GG 1.0(ref.)
AA 12.39 5.35(2.10–13.63) 0
GA 6.17 3.05(1.27–7.37) 0.013
Dominance GG 1.0(ref.)
AA+GA 8.76 3.63(1.55–8.51) 0.003
Recessive GG+GA 1.0(ref.)
AA 8.35 2.35(1.32–4.18) 0.004
rs10936601 Codominance TT 1.0(ref.)
CC 6.58 3.07(1.30–7.23) 0.01
TC 10.81 3.01(1.56–5.79) 0.001
Dominance TT 1.0(ref.)
CC+TC 11.91 3.02(1.61–5.66) 0.001
Recessive TT+TC 1.0(ref.)
CC 0.16
rs7726159 Codominance CC 1.0(ref.)
AA 6.82 2.69(1.28–5.65) 0.009
CA 0.68 1.32(0.69–2.53) 0.409
Dominance CC 1.0(ref.)
AA + CA 0.12
Recessive CC + CA 1.0(ref.)
AA 4.47 1.96(1.05–3.65) 0.034

Adjusted with systolic and diastolic blood pressure, aspartate aminotransferase, albumin, Serum creatinine, blood urea nitrogen, uric acid, cystatinC, serum potassium, serum sodium, hemoglobin, D-Dimer, NT-proBNP, pulmonary artery systolic pressure, and comorbidity of renal dysfunction.