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. 2021 Mar 9;12:639857. doi: 10.3389/fphys.2021.639857

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Copyright © 2021 Zhang, Chen, Dong and Xu.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Cyclopiazonic acid induction of mesenteric arterial relaxation through endothelium-dependent hyperpolarization. (A) Summary data showing the CRC, R_max, and EC₅₀ of CPA-induced vasorelaxation in mesenteric arteries in the absence (control, n = 6) or the presence of either 100 μM Nω-nitro-L-arginine (L-NNA; n = 6) or 10 μM indomethacin (INDO; n = 6). (B) Summary data showing the CRC, R_max, and EC₅₀ of CPA-induced vasorelaxation in the presence of L-NNA + INDO (n = 6), L-NNA + INDO (&) + 0.3 μM SKA-31 (n = 6), or L-NNA + INDO (&) + 10 mM tetraethylammonium chloride (TEA; n = 6). Data were expressed as percentage of NE (5 μM)-induced vasoconstriction and shown as means ± SEM. ^*p < 0.05, ^****p < 0.0001, and ns, no significance.