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. 2021 Mar 22;11:6570. doi: 10.1038/s41598-021-86083-8

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Pearson Correlation Analysis for plasma LPS levels and plasma CRP levels in different causes of stroke, TIA, and controls. The plasma LPS levels correlated with plasma CRP levels in controls (A), TIA (B), SVO stroke (C), LAA stroke (D), ICH (E), CE stroke (F) and for all subjects (G). Note that the intercepts of LPS trendlines on Y axis (CRP levels) in control, TIA, SVO stroke, LAA stroke, ICH stroke and CE strokes were − 0.8, − 1.6, 0.5, − 0.2, − 1.6 and 2.9 µg/ml, respectively (A–F). The intercept of LPS trendline on Y axis (CRP levels) for all 205 subjects was + 0.3 as indicated in (G). LPS lipopolysaccharide, CRP C-reactive protein, CE cardioembolic, LAA large artery atherosclerosis, SVO small-vessel occlusion, ICH intracerebral hemorrhagic, TIA transient ischemic attack.