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. 2021 Mar 23;54:11. doi: 10.1186/s40659-021-00335-5

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Oxidative (ox)-LDL(Ox-LDL) inhibited circRSF1 expression in HUVECs. a The graph of genomic loci of RSF1 gene and circRSF1. b The effect of actinomycin D (ActD) on cells was detected by qRT-PCR, and the abundances of circRSF1 and RSF1 mRNA were detected at specified time points. c RNase R digestion test was used to test the stability of circRSF1. After RNase R treatment, the expression of circRSF1 was more stable than RSF1 mRNA. d The expression of circRSF1 in nuclear and cytoplasmic of HUVECs e the expression of circRSF1 was detected in Control and ox-LDL of HUVECs. Each experiment was performed in triplicate *p < 0.05