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. 2020 Mar 18;26(2):e12896. doi: 10.1111/adb.12896

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© 2020 The Authors. Addiction Biology published by John Wiley & Sons Ltd on behalf of Society for the Study of Addiction

This is an open access article under the terms of the http://creativecommons.org/licenses/by/4.0/ License, which permits use, distribution and reproduction in any medium, provided the original work is properly cited.

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Interaction between serotonin transporter (SERT) deletion and cocaine self‐administration on the NMDA receptor obligatory subunit 1 GluN1 in the infralimbic (ILc) and prelimbic (PLc) cortices. Protein levels of GluN1 in the homogenate and postsynaptic density of ILc (Panel A,B) and PLc (Panel C,D) are expressed as percentages of SERT^+/+‐naive rats. Below the graphs, representative immunoblots are shown for GluN1 (120 kDa) receptor in the homogenate and postsynaptic density of ILc (Panel A,B) and PLc (Panel C,D), respectively. Histograms represent the mean ± SEM of five to six rats per group. ^* p < 0.05, ^** p < 0.01 versus SERT^+/+ naive; ^## p < 0.01 versus SERT^+/+ ShA; ^§ p < 0.05, ^§§§ p < 0.001 versus SERT^−/− naïve; ^£££ p < 0.001 versus SERT^−/− LgA (two‐way ANOVA followed by Fisher's least significant difference [LSD] multiple comparisons test)