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. 2021 Jan 7;130(3):853–864. doi: 10.1152/japplphysiol.00576.2020

Figure 5.

Figure 5.

Chronic exercise prevents to the onset of cardiomyopathy in the K/BxN mouse. A: heart cross-sectional morphology in sedentary wild-type (Wt), exercised wild-type, sedentary K/BxN, and exercised K/BxN mice. B: heart weight and heart weight relative to body weight. C: in sedentary wild-type, exercised wild-type, sedentary K/BxN, and exercised K/BxN mice. D: gene expression analysis related to heart failure and E: exercise remodeling. (For B–E, n = 10 male mice/group; ANOVA, *P < 0.05 compared to sedentary wild-type mice). Data are presented as means ± SE. Ex, exercised; Sed, sedentary; PGC, PPARG coactivator; CAV, caveolin; CEPBP, CCAAT enhancer binding protein; CITED, CREB-binding protein/p300-interacting transactivator with Asp/Glu-rich C-terminal domain; MYH, myosin heavy chain; ACTA, actin alpha; BNP, brain natriuretic peptide.