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. 2021 Mar 4:jeab042. doi: 10.1093/ehjci/jeab042

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Published on behalf of the European Society of Cardiology. All rights reserved. © The Author(s) 2021. For permissions, please email: journals.permissions@oup.com.

This article is made available via the PMC Open Access Subset for unrestricted re-use and analyses in any form or by any means with acknowledgement of the original source. These permissions are granted for the duration of the COVID-19 pandemic or until permissions are revoked in writing. Upon expiration of these permissions, PMC is granted a perpetual license to make this article available via PMC and Europe PMC, consistent with existing copyright protections.

This article is published and distributed under the terms of the Oxford University Press, Standard Journals Publication Model (https://academic.oup.com/journals/pages/open_access/funder_policies/chorus/standard_publication_model)

PMC Copyright notice

Three cases of CMR measurements included T2-STIR, Native T1 mapping, Post-contrast T1 (post T1), ECV, T2 mapping, and PSIR. The upper row (A–F) represents a healthy participant. The figures in the second row are of a patient (G–L) confirmed with COVID-19 and had a slightly increased TnI (0.08 ng/mL). The anterior wall displayed transmural hyperintensity. T1, T2, and ECV also displayed diffuse hyperintensity. The global T1, T2, and ECV were higher than the normal group, although the differences of PSIR were not significant. The third patient (M–R) with COVID-19 (ECG: T-wave depression, short PR interval, TnI: 0.14 ng/mL, LDH: 302 mmol/L) displayed typical T2-STIR and LGE hyperintensity in the sub-epicardium of the left ventricular inferior and inferior-lateral wall of the basal and middle parts.