Table 2.
BPA and BPS liver concentrations at different exposure times and exposure doses.
| Exposure time | Treatment | Dose | Conc. in liver |
|---|---|---|---|
| mg/kg | ng/g | ||
| 2 weeks | C | 0 | 0.02 ± 0.01a |
| n | 10 | ||
| BPA L | 2 | 27.2 ± 17.7b | |
| n | 10 | ||
| BPA H | 20 | 42.2 ± 23.1c | |
| N | 9 | ||
| BPS L | 2 | 25.3 ± 11.1b | |
| n | 10 | ||
| BPS H | 20 | 28.5 ± 21.8b | |
| n | 10 | ||
| 8 weeks | C | 0 | 0.02 ± 0.17a |
| n | 6 | ||
| BPA L | 2 | 37.6 ± 11.7b | |
| n | 10 | ||
| BPA H | 20 | 45.8 ± 12.7c | |
| n | 10 | ||
| BPS L | 2 | 33.4 ± 10.0b | |
| n | 7 | ||
| BPS H | 20 | 42.9 ± 15.3c | |
| n | 10 |
Treatments were performed at different concentrations (L = 2 mg/kg; H = 20 mg/kg) of BPA and BPS, at two different times of exposure (2 weeks and 8 weeks). Concentration in liver (conc. in liver) at the time point of sampling is indicated in ng/g (wet weight). C, control group. Letters indicate statistically significant difference with respect to control within each group (MRT p < 0.05). n, number of individuals. Assessments were performed on 49 individuals for the 2 weeks’ and 43 individuals for the 8 weeks’ exposure. Data are shown as mean ± standard deviation.