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. 2021 Feb 26;13(5):7538–7548. doi: 10.18632/aging.202616

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Copyright: © 2021 Zhou et al.

This is an open access article distributed under the terms of the Creative Commons Attribution License (CC BY 3.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

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SMCs specific SETD2 deficient ameliorates right ventricular function in a hypoxia-induced mouse model of PAH. Echocardiography was used to measure the normalized cardiac cycle length measuring pulmonary acceleration time (PAAT/CL), RV end diastolic dimension (RVEDD), RV diastolic contraction size (RVESD), RV end diastolic volume (RVEDV), RV diastolic volume contraction (RVESV), RV cardiac output (RVCO), RV wall thickness (RVWT), the thickness of interventricular septum (IVS), RV ejection fraction (RVEF) and RV shortening fraction (RVFS). All data were presented as Mean±SD (n=5). *P<0.05, **P<0.01 vs. corresponding group.