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Reactive oxygen species (ROS) are increased in obese people, favoring the oxidized form of HSA (HNA), which in turns, generates an increase in proinflammatory cytokines. Here, we hypothesized (discontinuous lines) that the HNA, particularly oxidized at Cys34, may be generated as a result of obesity, promoting the progression of inflammatory and degenerative injuries, which are crucial for the progression of chronic kidney disease.
