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. 2021 Mar 30;11:7197. doi: 10.1038/s41598-021-85477-y

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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The absence of Myo1g limits the ability of B lymphocytes to generate morphological changes during migration. Schematic illustration of the role of Myo1g during activated B lymphocytes migration. In absence of Myo1g activated B lymphocytes have a lower capacity to exert changes in their morphology and lower surface expression of adhesion molecules. These changes increase their speed of movement, reducing the interaction between the B lymphocyte and the endothelium, directly affecting homing of B lymphocytes towards the inguinal lymph node. Created with BioRender.com.