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. 2020 Jun 20;37(2):261–275. doi: 10.1007/s10565-020-09542-4

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — a Cell cycle distribution after pretreated with MSM EC cells before DOX exposure. The cell cycle distribution is expressed as % of gated cells. p < 0.05 was considered statistically significant. *p < 0.05, **p < 0.01, ***p < 0.001 as compared to Cnt. #p < 0.05, ##p < 0.01, ###p < 0.001 as compared to MSM treatment. MSM methylsulfonylmethane, DOX doxorubicin, Cnt control. b Expression of CDKN1A after DOX, MSM, and MSM + DOX treatment in EC cells. The expression of CDKN1A was evaluated in RTqPCR. The results are expressed as a relative expression. p < 0.05 was considered statistically significant. *p < 0.05, **p < 0.01, ***p < 0.001 as compared to Cnt. #p < 0.05, ###p < 0.001 as compared to MSM treatment. MSM methylsulfonylmethane, DOX doxorubicin, Cnt control