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. 2021 Mar 15;13(3):1400–1410.

Figure 1.

Figure 1

Cell changes and passive release of HMGB-1 after inducing necrosis by H2O2 treatment in ectopic HESCs. Oxidative stress caused cell proliferation, although not statistically significant (A), decrease of cell viability (B), and increase of HMGB-1 release (C) in ectopic HESCs. *P < 0.05 compared to 0 μM H2O2 treatment. #P < 0.05 compared to 50 μM H2O2 treatment.