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. 2021 Mar 19;12:629379. doi: 10.3389/fphar.2021.629379

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Copyright © 2021 Duan, Zhang, Yang, Su, Zhou, Lo, Cai, Qiao, Li, Huang, Wang, Mo and Wang.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Proposed mechanistic model of baicalin protects against hemin-induced ferroptosis in ICH. Our findings demonstrated that baicalin effectively enhances the expression levels of GPX4 and SLC7A11, and inhibits the accumulation of lipid ROS and eventually protects against hemin-induced ferroptosis and ICH-induced brain injury.