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Functional model of the IL-6 involved in restraining EMT in OC progression after downregulation of RIPK4 expression. RIPK4-silenced OC cells hypoactivated IL-6 signalling. Less STAT3 was phosphorylated, and the expression of SOCS1/3 was reduced. Consequently, the RIPK4-based IL-6 network is a determinant of EMT in OC.
