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. Author manuscript; available in PMC: 2021 Apr 6.
Published in final edited form as: Neuroimage. 2021 Jan 21;229:117737. doi: 10.1016/j.neuroimage.2021.117737

Fig. 1.

Fig. 1.

Definitions, hypotheses and study setup, A: Illustration of high and low redundancy networks under disruption, B: Different trajectories of hypothesized variation of functional network redundancy over the human lifespan, Hypothesis 1 follows the pattern of accumulation and later-age decline, Hypothesis 2 suggests early development followed by slow decline, Hypothesis 3 takes the course of accrual similar to 1, but does not lead to old-age decline, C: Schematic showing the hypothesized framework for age-cognition-redundancy relationship. We hypothesize that the established association between age and cognition (indicated by arrow), will be mediated by redundancy (dashed arrows) operating as a mechanism of reserve in this context, D: Age distribution of the participants in the study.