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. 2021 Apr 6;22:117. doi: 10.1186/s12882-021-02309-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 1 — Slit lamp examination shows diffuse intracorneal crystalline deposits (a); immunohistochemistry staining for kappa light chains shows positivity of the crystals in the podocytes (b) with lambda negativity (c); glomerular staining with methylene-azure-blue stains light chain crystals strongly blue with a crystalline appearance (d); tubular staining with methylene-azure-blue shows scarce crystalline inclusions in the cytoplasm of proximal tubular epithelial cells (e); transmission electron microscopy confirms these crystalline inclusions diagnostic for minimal proximal light chain tubulopathy as intralysosomal (f)