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. 2020 Jul 30;46(3):569–578. doi: 10.1038/s41386-020-0781-1

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — a Experimental timeline. b Average GluA1 surface expression in obesity-prone (OP) and obesity-resistant (OR) rats. GluA1 surface expression increased following 2 weeks of junk-food deprivation in obesity-prone, but not obesity-resistant rats. c Average GluA2 surface expression in obesity-prone and obesity-resistant rats. GluA2 surface expression was not altered by junk-food in either group. All data shown as mean ± SEM unless otherwise noted. *p < 0.05 obesity-prone chow vs. p < 0.05 junk- food.