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. 2021 Jan 20;28(4):1149–1158. doi: 10.1038/s41418-020-00729-0

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Deletion or low expression of either of the MPC subunits in cardiac tissue leads to downregulation of the other subunit without altering its transcript levels. This indicates that the turnover of MPC complex might be controlled by ubiquitination or autophagy processes happening at the protein level.