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ECMR mediated activation of EnNaC in the pathogenesis of vascular stiffness in hyperaldosteronism. ECMR, endothelial cell; EnNaC, endothelial cell sodium channel; eNOS endothelial cell nitric oxide synthase; NO, nitric oxide; SGK1, serum glucocorticoid kinase; VSMC, vascular smooth muscle cells
