Table 3.
Reference | Sample size and subjects included in the study | Main findings |
---|---|---|
Tadic et al10 | 58 individuals with optimal blood pressure, 57 individuals with high‐normal blood pressure, and 59 recently diagnosed untreated hypertensive patients of similar age and sex distribution. | RV GLS gradually reduced from controls, across prehypertensive individuals, to hypertensive patients. RV wall thickness simultaneously associated with global RV and right atrial longitudinal strain, and RV diastolic function. |
Tadic et al37 | 153 untreated patients (50 normal blood pressure, 48 WCH, 55 hypertensive subjects) | Results showed that LV and RV GLS gradually deteriorated from controls to patients with sustained hypertension. Endocardial RV longitudinal strain was lower in WCH and hypertensive patients than in controls. Mid‐myocardial RV longitudinal strain was decreased in hypertensive patients compared with the other 2 groups, whereas subepicardial RV longitudinal strain was similar between the observed groups. 24‐h systolic blood pressure was associated with subendocardial RV strain. |
Tadic et al38 | 186 untreated subjects (56 normal blood pressure, 60 MH, 70 hypertensive subjects) | Global and free‐wall RV longitudinal strains were significantly lower in MH and sustained hypertensive patients comparing with controls. Endocardial and mid‐myocardial RV longitudinal and circumferential strains were significantly reduced in MH and hypertensive patients. There was no difference between MH and subjects with sustained hypertension. |
GLS, global longitudinal strain; LV, left ventricle; MH, masked hypertension; RV, right ventricle; WCH, white coat hypertension.