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. 2021 Mar;9(5):364. doi: 10.21037/atm-20-5136

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Correlation of pathological homologous recombination repair (HRR) gene mutations with residual disease and platinum sensitivity. Data among the three groups were compared with Pearson chi-square tests with P values. (A) Residual disease status among three groups. The R0 rates in the wild-type, germline and somatic homologous recombination repair (HRR) mutated groups were 48.3%, 28.6% and 0%, respectively (P=0.233). (B) Response to platinum treatment in each group. There were no statistically differences among three groups (wild-type vs. germline vs. somatic, 62.1 vs. 42.9 vs. 50%, respectively, P=0.851).