Relative mRNA expression levels of selected CLEAR network genes in HCT116 cells treated as indicated for 16 h (NEN (N) 1.2 µM, domperidone (Domp, D) 30 µM, imipramine (Imi), desipramine (Desi), amitriptyline (Ami), each 30 µM, clomipramine (Clomi) 20 µM).
Immunoblot analysis of whole cell lysate (WL), cytoplasmic (Cyto) and nuclear (Nu) fractions of HCT116 cells using antibodies against the indicated proteins upon treatment of the cells as indicated for 16 h (Control (Ctrl), NEN (N) 1.2 µM, domperidone (D) 30 µM). GAPDH and acetyl‐histone H3 (AC‐H3) served as markers for cytoplasmic and nuclear fraction, respectively.
Immunoblot analysis and quantification (mean ± SD) of LC3‐II/LC3‐I ratios and P62 in HCT116 cells. Cultured cells were treated with DMSO (Control), NEN or NEN and imipramine (Imi) as indicated (NEN, 1.2 µM; Imi, 30 µM) for 16 h. Protein band signal intensities were normalized to vinculin (loading control), and the ratios LC3‐II bands LC3‐I (N = 3) and p62 levels (N = 3) were analyzed by one‐way ANOVA with Tukey post hoc test. *P < 0.05; **P < 0.01; ***P < 0.001; ****P < 0.0001.
Immunoblot analysis and quantification (mean ± SD) of LC3‐II/LC3‐I ratios and P62 in HCT116 cells. Cultured cells were treated with DMSO (Control), NEN or NEN and amitriptyline (Ami) as indicated (NEN, 1.2 µM; Imi, 30 µM) for 16 h. Protein band signal intensities were normalized to vinculin (loading control), and the ratios LC3‐II bands LC3‐I (N = 3) were analyzed by one‐way ANOVA with Tukey post hoc test. **P < 0.01; ***P < 0.001; ****P < 0.0001.