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. 2021 Apr 9;12(4):385. doi: 10.1038/s41419-021-03671-2

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons license, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons license, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons license and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this license, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 6 — Western blot analysis of p-AMPK, p62, and LC3II in VSMCs. A Treated with DMSO or 30 μM CMLD-2 for 24 h (n = 5). B Transfected with CTR or HuR siRNA for 48 h (n = 5). C Infected with ad-GFP or ad-HuR (n = 5), and D treated with PBS or 0.5 μg/μl recombinant HuR protein for 48 h (n = 5). E Western blot analysis of p-AMPK, p62, and LC3II in aortas from CTR and HuR^SMKO mice (n = 5).