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Schematic representation of the experimental settings to determine the role of the p65 pathway in the release of small extracellular vesicles (sEV). sEV were isolated from (A) cellular senescence models (DNA damage-induced senescence (DDIS) and therapy-induced senescence (TIS)) and (B) proinflammatory activation (PA) in mesenchymal stem cells (MSCs) after the treatment with pharmacological inhibitors (JSH-23, MG-132 and curcumin) during the induction.
