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. 2021 Mar;9(6):481. doi: 10.21037/atm-21-621

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2021 Annals of Translational Medicine. All rights reserved.

Open Access Statement: This is an Open Access article distributed in accordance with the Creative Commons Attribution-NonCommercial-NoDerivs 4.0 International License (CC BY-NC-ND 4.0), which permits the non-commercial replication and distribution of the article with the strict proviso that no changes or edits are made and the original work is properly cited (including links to both the formal publication through the relevant DOI and the license). See: https://creativecommons.org/licenses/by-nc-nd/4.0.

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Effects of ANG II and NE on the AMPK/PGC-1α/NRF-2 pathway. Rats were euthanized 24 h after the start of the experiment. (A,B,C) A Western blotting analysis was performed to determine protein levels of PGC-1α, NRF-2, p-AMPK, and AMPK in the rat kidneys. β-actin was used as an internal control. Data are expressed as the mean ± SEM. n=8 in the control or sham, n=5 in the CLP, n=6 in the CLP + ANG II, n=6 in the CLP + NE groups. PGC-1α, peroxisome proliferator-activated receptor-γ coactivator-1-α; NRF2, nuclear respiratory factor; AMPK, AMP-activated protein kinase; p-AMPK, phosphorylated AMPK.