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. 2021 Apr 12;9(4):E552–E561. doi: 10.1055/a-1352-2709

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The Author(s). This is an open access article published by Thieme under the terms of the Creative Commons Attribution-NonDerivative-NonCommercial License, permitting copying and reproduction so long as the original work is given appropriate credit. Contents may not be used for commercial purposes, or adapted, remixed, transformed or built upon. (https://creativecommons.org/licenses/by-nc-nd/4.0/)

This is an open-access article distributed under the terms of the Creative Commons Attribution-NonCommercial-NoDerivatives License, which permits unrestricted reproduction and distribution, for non-commercial purposes only; and use and reproduction, but not distribution, of adapted material for non-commercial purposes only, provided the original work is properly cited.

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Fig. 5 — Increased expression of vascular endothelial growth factor in dysplastic epithelia. a Representative images of p53 and vascular endothelial growth factor (VEGF) expression in non-neoplastic (Non-n), flat-type low-grade dysplasia (LGD) and flat-type high-grade dysplasia (HGD) (Case #4). Note that flat-type LGD and HGD epithelia expressed increased expression of VEGF compared to the surrounding non-neoplastic epithelia. Moreover, the pattern of VEGF expression in dysplastic epithelia was similar to that of p53. Scale bars, 200 μm. b Schematic presentation showing the association among epithelial p53 expression, VEGF expression and stromal vascular alteration.