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. 2021 Apr 12;11:7937. doi: 10.1038/s41598-021-87199-7

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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CCL11 induced the migration of THP-1 cells. (A) CCL11-stimulated THP-1 cells were significantly more efficient at migration than unstimulated cells (n = 4 experiments). SF synovial fluid. (B) CCL11 siRNA treatment decreased the secretion of CCL11 in TNF-α-treated RA FLS-conditioned medium (n = 3 patients). (C) CCL11 siRNA-transfected and TNF-α-stimulated RA FLS-conditioned medium reduced THP-1 migration compared to control siRNA-transfected and TNF-α-stimulated RA FLS-conditioned medium (n = 4 patients). The data are expressed as the mean ± SEM. *p < 0.05 was significant.