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. 2020 Jul 31;14(2):256–270. doi: 10.1007/s12265-020-10057-w

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© The Author(s) 2020

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 3 — Effects of acute exercise on glucose uptake and insulin signaling. Acute exercise evokes an increase of glucose uptake which is time-dependently regulated by insulin-independent and insulin-dependent mechanisms. The exercise-induced enhanced insulin sensitivity is mediated by different pathways that work in parallel with the canonical insulin signaling cascade. AMPK: AMP-activated protein kinase; AS 160: AS 160 protein; GLUT4: insulin-responsive glucose transporter; IRS: insulin receptor substrates; PI3K: phosphoinositide 3-kinase