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. 2021 Apr 13;11:8002. doi: 10.1038/s41598-021-87061-w

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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L3 and D3-derivatives activate a LXR-response element (LXRE)-driven reporter in CHO cells (A,C) and human HaCaT keratinocytes (B,D). Representative dose response curves are in (A) and (B), while a summary of assays performed with 10^–7 M ligands is presented for each experiment separately for CHO cells (C) and HaCaT keratinocytes (D). Data are presented as means ± SE, (n = number of assays). Analysis was done using one-way ANOVA for dose responses and the t-test: *p < 0.05, **p < 0.01, ***p < 0.001 or ****p < 0.0001 versus control (ethanol).