Skip to main content
. 2021 Mar 31;9:631329. doi: 10.3389/fped.2021.631329

Table 2.

The main altered genes described in the monogenic forms of SLE and corresponding phenotype.

Locus Gene location Protein Inheritance Pathway Phenotype
C1QA
C1QB
C1QC
1p36.12 C1q AR Complement—Classic pathway Nephritis, CNS involvement, photosensibility, arthritis, infectious susceptibility
C1R 12p13.31 C1r AR Complement—Classic pathway Poliarthicular arthritis, upper and lower respiratory tract infections
C1S 12p13.31 C1s AR Complement—Classic pathway Hashimoto's thyroiditis, autoimmune epatitis
C2 6p21.33 C2 AR Complement—Classic pathway Arthritis, malar rash, discoid rash, and photosensibility. Rare neurological and renal involvement
C4A-B 6p21.33 C4 AR Complement—Classic pathway Multiorgan involvement with severe nephritis and photosensibility
C3 6p21.33 C4 AR Complement—Classic and no classic pathway Complement deficiencies with upper and lower respiratory tract infection, SLE in a minority of affected individuals
DNASE 1 16p13.3 DNASE1 AD Extracellular acid nucleic degradation Sjogren syndrome, high level of antinucleosomal autoantibodies
DNASE1L3 3p14.3 DNASE1L3 AR Extracellular acid nucleic degradation Early onset, nephritis, ANCA positive hypocomplementemic urticarial vasculitis syndrome (HUVS)
ACP5 2q24.2 TRAP AD Type-I IFN—regulation of Opn Cytopenia (also implied in SMS)
IFIHI 2q24.3 MDA-5 AD Type-I IFN Cytosolic sensor for dsRNA IgA deficiency, mild lower limb
Spasticity (also implied in SMS and AGS)
TREX1 3p21.31 TREX1 AD Type-I IFN degradation of intracellular ds-ss DNA FCL (also implied in AGS)
SAMHD1 20q11.23 SAMHD1 AR Type I IFN cytoplasmic ssRNA/DNA sensor FCL (also implied in AGS)
PRKCD 3p21.2 PRKCD AR Self-tolerance-B-cell survival and proliferation Renal involvement, CNS vasculitis, lymphoproliferative syndromes
RAG2 11p12 RAG2 AR/AD Self-tolerance Immune-mediated cytopenias to localized destructive vasculitis
TNFRSF6 10q23.31 CD95 AR Self-tolerance- apoptosis Marked lymphadenopathy

AD, autosomal dominant; AR, autosomal recessive; ACP5-TRAP, Acid Phosphatase 5 Tartrate Resistant; IFIH1, IFN-induced helicase C domain-containing protein 1; MDA5, melanoma differentiation-associated protein 5; TREX1, DNA 3′ repair exonuclease; SAMHD1, deoxynucleoside triphosphate triphosphohydrolase SAM domain and HD domain 1; PRKCD, Protein Kinase C Delta; RAG 2, recombination-activating genes 2; TNFRSF6, Truncated tumor necrosis factor receptor superfamily member 6; FLC, familial chilblain lupus.