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. 2021 Mar 25;9:644901. doi: 10.3389/fcell.2021.644901

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Copyright © 2021 Zhu, Zhang, Huang, Lin, Fan and Ni.

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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In vivo effect of TRIM26 overexpression on CCl₄-induced liver fibrosis. Mice liver fibrosis model was established by CCl₄ induction. Recombinant adenovirus Vector or oeTRIM26 was injected into the mice through tail vein. (A) Histological examination (magnification at 200×) of liver tissues by hematoxylin and eosin, Masson’s trichrome, and Sirius red staining. (B) The serum levels of alanine transaminase (ALT) and aspartate transaminase (AST). (C) The serum levels of hydroxyproline (Hyp). (D) Proteins expressions of TRIM26 and SLC7A11 in liver tissues. (E) Protein expressions of α-SMA and collagen I in liver tissues. **p < 0.01; ***P < 0.001 vs. Vehicle; ^#P < 0.05, ^##P < 0.01, and ^###P < 0.001 vs. CCl₄ + Vector.