Figure - PMC

Skip to main content

An official website of the United States government

Here's how you know

Here's how you know

Official websites use .gov
A .gov website belongs to an official government organization in the United States.

Secure .gov websites use HTTPS
A lock ( ) or https:// means you've safely connected to the .gov website. Share sensitive information only on official, secure websites.

View full-text article in PMC

. 2021 Apr 13;26:14. doi: 10.1186/s11658-021-00258-9

Search in PMC
Search in PubMed
View in NLM Catalog
Add to search

© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

PMC Copyright notice

Fig. 6 — CDKN2B-AS1 expression in normal and cancer states. a lncRNA expression in 27 normal tissues from GTEx Portal showed upregulation of CDKN2B-AS1 in colorectal tissue and salivary glands followed by neurons while being down-regulated in breast tissue. b Expression level of lncRNA in breast cancer tissues with different receptor status. c Subcellular localization of CDKN2B-AS1. d–f Analysis of 8925 breast cancer patients in the TCGA across 16 studies. d Expression level of CDKN2B-AS1 according to the gene copy number. e One hundred forty-six patients (2.1%) had a genetic alteration in the CDKN2B-AS1 gene (44 amplifications and 102 deep deletions). f Patients with altered CDKN2B-AS1 had poor survival