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. 2021 Apr 14;21:178. doi: 10.1186/s12872-021-01975-x

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 2 — 3D volume rendered reconstruction of the heart and coronary arteries showed high take off of the right coronary artery (a) and distal segment of the left anterior descending coronary artery and diagonal branch with a superficial intra-myocardial course (arrow) (b). Resting echocardiography showed focal septal wall motion abnormalities called septal buckling (arrow) at end systole (c) and early diastole (d). T2-weighted heart MRI image showed myocardial wall thinning and subendocardial myocardial oedema (arrow) in the mid-ventricular to apical anterior and septal regions (e). Late gadolinium enhancement imaging showed a high signal intensity in the same region (f)