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. 2021 Mar 31;11:644840. doi: 10.3389/fonc.2021.644840

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Copyright © 2021 Yan, He, Li, Liu and Gao

This is an open-access article distributed under the terms of the Creative Commons Attribution License (CC BY). The use, distribution or reproduction in other forums is permitted, provided the original author(s) and the copyright owner(s) are credited and that the original publication in this journal is cited, in accordance with accepted academic practice. No use, distribution or reproduction is permitted which does not comply with these terms.

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Effects of ACSM3 on the Integrin β1/AKT signaling pathway in ovarian cancer. (A) The protein expression of Integrin β1, p-AKT, AKT, cyclin D1, c-Myc, Vimentin, and E-cadherin in OV-90 cells following ACSM3 knockdown. (B) The protein expression of Integrin β1, p-AKT, AKT, cyclin D1, c-Myc, Vimentin, and E-cadherin in A2780 cells following ACSM3 overexpression. Measurement data were expressed as mean ± SD of three independent experiments. **P < 0.01 versus Lv-shRNA-NC; ^##P < 0.01 versus Lv-Vector.