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GEVs were secreted by G. duodenalis and entered into host cells triggering inflammatory response by recognizing extracellular TLR2 and intracellular NLRP3. NLRP3 inflammasome was activated, mediated IL-1β release and controlled cytokines secretion. This process could be completely reversed by CA-074 methyl ester/zVAD-fmk or partial reversed by C29/Glibenclimide/Ac-YVAD-CHO.
