A) Original traces of echocardiographic M-mode acquisitions
(parasternal long axis view) in CASK-CTRL or CASK-KO mice before TAC, and at two
weeks after TAC operation. B) Mean data of left ventricular
ejection fraction (EF). Data follows normally distribution (Shapiro-Wilk-test,
*two-way repeated measured mixed-effects analysis p for TAC=0.0459 with
Holm-Sidak post-test (p values in graph, n=mice). C) Kaplan-Meier
survival analysis for CASK-CTRL, CASK-Flox and CASK-KO mice upon TAC. Data were
tested by Log-rank (Mantel-Cox) with p in graph. n=mice at risk. D)
Mean densitometric expression of CASK (normalized to GAPDH, western blot) in
left ventricular homogenates of mice after TAC (vs. Sham-operated mice). Data
are normally distributed (Shapiro-Wilk-test) and tested by unpaired t-test (p in
graph, n=mice. E) Original registrations (western blot) and mean
densitometric data for CaMKII expression and T286 autophosphorylation in
ventricular homogenates from CASK-CTRL and KO mice 2 weeks (2w) after TAC. Left
panel: Data are not normally distributed (Shapiro-Wilk) and tested by
*Kruskall-Wallis p=0.0089. Multiple comparison were done by two-stage linear
step-up procedure of Benjamini, Krieger and Yekutieli (p in graph, n=mice. Right
panel: Data are normally distributed (Shapiro-Wilk-test) and tested by *ANOVA
p=0.0132 with Holm-Sidak post-test (p in graph, n=mice). F) Mean
densitometric data for T305-autophosphorylation shows that compared to
CASK-CTRL, T305 autophosphorylation was significantly reduced in CASK KO upon
TAC. Data are normally distributed (Shapiro-Wilk-test) and compared by unpaired
t-test (p in graph, n=mice). G) Mean densitometric analysis of
CaMKII-oxidation relative to CaMKII expression indicates enhanced CaMKII
oxidation in CASK KO upon TAC. Data are normally distributed (Shapiro-Wilk-test)
and compared by unpaired t-test (p in graph, n=mice. Label
information: CASK-CTRL = CTRL, CASK-Flox = Flox, CASK-KO =
KO