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. 2020 Apr 8;15(2):143–154. doi: 10.31616/asj.2019.0285

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Copyright © 2021 by Korean Society of Spine Surgery

This is an Open Access article distributed under the terms of the Creative Commons Attribution Non-Commercial License (http://creativecommons.org/licenses/by-nc/4.0/) which permits unrestricted non-commercial use, distribution, and reproduction in any medium, provided the original work is properly cited.

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Fig. 4. — Gene expression of pro-inflammatories (TNF-α, IL-1β, and IL-6), metalloproteases (MMP-2, MMP-3; ADAMTS-5), anti-inflammatories (IL-2 and IL-10), cell death marker (CYT-C), stress-cum-repair marker (IGF1R), collagen 9A1, and endogenous control (GAPDH) in all experimental groups. TNF, tumor necrosis factor; IL, interleukin; MMPs, matrix metalloproteinases; CYT-C, cytochrome c; IGF1R, insulin-like growth factor 1 receptor; COL9A1, collagen 9 alpha 1; GAPDH, glyceraldehyde 3-phosphate dehydrogenas.