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. 2021 Feb 11;6(1):e120. doi: 10.1097/j.pbj.0000000000000120

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Copyright © 2021 The Authors. Published by Wolters Kluwer Health, Inc. on behalf of PBJ-Associação Porto Biomedical/Porto Biomedical Society. All rights reserved.

This is an open access article distributed under the terms of the Creative Commons Attribution-Non Commercial-No Derivatives License 4.0 (CCBY-NC-ND), where it is permissible to download and share the work provided it is properly cited. The work cannot be changed in any way or used commercially without permission from the journal. http://creativecommons.org/licenses/by-nc-nd/4.0

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Metabolic changes in cellular senescence. Activation of the key signalling pathways that act as metabolic switches to regulate senescence. ADP = adenosine dinucleotide; AMP = adenosine mononucleotide; AMPK = AMP-activated protein kinase; ATP = adenosine trinucleotide; HuR = Hu antigen receptor; mRNA = messenger RNA; mTORC1 = mammalian target of rapamycin complex 1; NAD+ = oxidized nicotinamide adenine dinucleotide; NADH reduced nicotinamide adenine dinucleotide; p16 = cyclin-dependent kinase inhibitor 16; p21 = cyclin-dependent kinase inhibitor 21; p53 = tumour suppressor p53 protein; pRB = tumour protein of the retinoblastoma.