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. 2021 Apr 8;11(2):120–124. doi: 10.4103/ijabmr.IJABMR_579_20

Table 2.

Histopathological findings

Site of tissue Microscopic findings
Lungs Intraalveolar edema, congestion of alveolar septal capillaries and larger vessels, septal perivascular lymphocytic infiltrate and peribronchiolar lymphocytic infiltrate, focal interalveolar exudates of macrophages admixed with lymphocytes scattered small epithelioid cell granulomas with foreign body and Langerhans types of giant cells and peripheral cuffing by lymphocytes in predominantly perivascular distribution, however, ZN stain for AFB was negative [Figure 2]
Liver Congestion, mild chronic inflammatory infiltrate in some portal tract, and occasional lymphocytic aggregate adjacent to central vein [Figure 3a and b]
Spleen Congestion [Figure 3c]
Brain No pathological changes observed
Kidney Congestion, occasional tiny focus of chronic inflammatory cells aggregate [Figure 3d]
Heart No pathological changes observed
Tracheal tissues Sparse submucosa and perivascular lymphocytic infiltrate mucosal epithelium shows partial autolysis [Figure 4a and b]
Salivary gland The salivary gland shows focal sparse perivascular lymphocytic infiltrate [Figure 5a]
Epiglottis Sparse submucosal lymphocytic infiltrate [Figure 5b and c]
Upper Oesophagus Shows focal sparse perivascular lymphocytic infiltrate [Figure 5d]
Tongue Follicular lymphoid hyperplasia, on its posterior aspect [Figure 4c]
Pharyngeal tissues Edema, sparse sub mucosal lymphocytic infiltrate, and few submucosal lymphoid follicles [Figure 4d]
Tonsils Reactive follicular hyperplasia