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. 2021 Jan 27;29(5):280–287. doi: 10.1007/s12471-021-01538-x

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© The Author(s) 2021

Open Access This article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article’s Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article’s Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/.

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Fig. 1 — After combining an extensive clinical background with non-invasive measurements and cardiac tissue, we will look for: (1) the combination of genetic data of blood and tissue in combination with data on structural remodelling, which would give insight into the different genes involved in structural remodelling; (2) the possible association or predisposing role of structural remodelling in atrial fibrillation (AF) and AF development; (3) the difference in genetic expression in association with different types of AF and the genetic markers for predisposition for postoperative atrial fibrillation (POAF) development. eQTLs expression quantitative trait loci, mRNA messenger RNA