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. 2021 Apr 23;15:24. doi: 10.1186/s40246-021-00321-8

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© The Author(s) 2021

Open AccessThis article is licensed under a Creative Commons Attribution 4.0 International License, which permits use, sharing, adaptation, distribution and reproduction in any medium or format, as long as you give appropriate credit to the original author(s) and the source, provide a link to the Creative Commons licence, and indicate if changes were made. The images or other third party material in this article are included in the article's Creative Commons licence, unless indicated otherwise in a credit line to the material. If material is not included in the article's Creative Commons licence and your intended use is not permitted by statutory regulation or exceeds the permitted use, you will need to obtain permission directly from the copyright holder. To view a copy of this licence, visit http://creativecommons.org/licenses/by/4.0/. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated in a credit line to the data.

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Fig. 3 — Silencing CAMP gene inhibited the proliferation of osteoarthritis articular cartilage cells and promoted their apoptosis. a The expression level of CAMP mRNA in Control, sh-NC, and sh-CAMP-transfected HC-OA cells was detected by qRT-PCR. b the expression level of CAMP protein in Control, sh-NC, and sh-CAMP-transfected HC-OA cell was detected by western blot. c The cell proliferation ability of Control, sh-NC, and sh-CAMP-transfected HC-OA cells was detected by CCK-8. d The apoptotic ability of Control, sh-NC, and sh-CAMP-transfected HC-OA cells was detected by flow cytometry. ^**p < 0.01, ^***p < 0.001, ^****p < 0.0001, compare with Control